In a joint collaborative battle Daisy Leung pubished a mechanism employed by NS1 of RSV to modulate gene transcription. We identified the critical binding partner of NS1 involved in this process.
Our collaborators from the Landolfo Lab discovered that HCMV infection triggers viral and host protein citrullination. We helped to characterize the consequences on IFIT1 antiviral function.
The Brinkmann Lab employs a multi-omic approach to identify novel antiviral roles of ZAP in decelerating HCMV infections. We bring in the proteomic component.
We helped our collaborators from the Hartmann and Imler labs find two cGAS-like receptors that regulate antiviral immunity in flies!
Our collaboration with the Protzer Lab helps identify a new anti-HBV host factor, ISG20!
Scientific community can use these deep and comprehensive data to understand COVID-19 molecular mechanisms.
In a joint collaborative battle Daisy Leung pubished a mechanism employed by NS1 of RSV to modulate gene transcription. We identified the critical binding partner of NS1 involved in this process.
Our collaborators from the Landolfo Lab discovered that HCMV infection triggers viral and host protein citrullination. We helped to characterize the consequences on IFIT1 antiviral function.
The Brinkmann Lab employs a multi-omic approach to identify novel antiviral roles of ZAP in decelerating HCMV infections. We bring in the proteomic component.
We helped our collaborators from the Hartmann and Imler labs find two cGAS-like receptors that regulate antiviral immunity in flies!
Our collaboration with the Protzer Lab helps identify a new anti-HBV host factor, ISG20!
Scientific community can use these deep and comprehensive data to understand COVID-19 molecular mechanisms.
Prof. Dr. Andreas Pichlmair
Immunopathology of Virus Infections Laboratory,
Institute of Virology,
Technical University of Munich
Schneckenburgerstr. 8,
D-81675 Munich, Germany
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